<?xml version="1.0" encoding="UTF-8"?><xml><records><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Shabina Komath Chenoly</style></author><author><style face="normal" font="default" size="100%">Vinodini NA</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Abelmoschus esculentus Ameliorates Stress-Induced Cognitive Dysfunction via Antioxidant and Neuroprotective Mechanisms in Mice</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Abelmoschus esculentus</style></keyword><keyword><style  face="normal" font="default" size="100%">Acetylcholine esterase</style></keyword><keyword><style  face="normal" font="default" size="100%">chronic stress</style></keyword><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Morris water maze</style></keyword><keyword><style  face="normal" font="default" size="100%">Superoxide dismutase</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2025</style></year><pub-dates><date><style  face="normal" font="default" size="100%">December 2025</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">17</style></volume><pages><style face="normal" font="default" size="100%">302-309</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Objective:&lt;/strong&gt; The objective of this study was to determine if there were any effect of Abelmoschus esculentus on chronic stress induced memory loss. &lt;strong&gt;Methods: &lt;/strong&gt;The Swiss Albino Mice (n=30) were divided into five groups, namely control, chronic stress (90 days), chronic stress and ethanolic extract of Abelmoschus esculentus(200mg/kg body weight), and Chronic stress followed by diazepam (2mg/kg body weight), Chronic stress followed by diazepam (2mg/kg body weight) and ethanolic extract of abelmoschus esculentus (200mg/kg body weight). At the end of the experimental period (90 days), animal blood was collected retro-orbitally to analyze the cortisol, Brain tissue was homogenized, and antioxidants and Acetylcholine esterase level was estimated. &lt;strong&gt;Results: &lt;/strong&gt;The Results of the data indicated that administering stress for a duration of 90 days led to a noticeable decline in spatial learning abilities in mice. Our findings indicate that combination of Abelmoschus esculentus and diazepam showed there was an significant improvement in retaining memory. &lt;strong&gt;Conclusion: &lt;/strong&gt;Study indicated that Abelmoschus esculentus shows promise as a viable treatment option for conditions of stress related memory impairment.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">6</style></issue><work-type><style face="normal" font="default" size="100%">Original Article</style></work-type><section><style face="normal" font="default" size="100%">302</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Shabina Komath Chenoly&lt;sup&gt;1&lt;/sup&gt;, Vinodini NA&lt;sup&gt;2*&lt;/sup&gt; &lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Associate Professor, Department of Physiology, Vydehi Institute of Medical Sciences &amp;amp; Research Center EPIP area, Bangalore, 5600066, INDIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Associate Professor, Department Of Physiology, Kasturba Medical college Mangalore, Manipal Academy of Higher Education, Manipal, INDIA.&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Anchana Babu</style></author><author><style face="normal" font="default" size="100%">Rekha D Kini</style></author><author><style face="normal" font="default" size="100%">Nayanatara Arun Kumar</style></author><author><style face="normal" font="default" size="100%">Megha Gokul</style></author><author><style face="normal" font="default" size="100%">Vandana Blossom</style></author><author><style face="normal" font="default" size="100%">Sreerag P</style></author><author><style face="normal" font="default" size="100%">Shymala Nayak</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Antioxidant and Neuroprotective Potential of Ashwagandha In Aluminum-Induced Toxicity</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Aluminum Chloride</style></keyword><keyword><style  face="normal" font="default" size="100%">Ashwagandha</style></keyword><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Oxidative stress</style></keyword><keyword><style  face="normal" font="default" size="100%">Reactive Oxygen Species</style></keyword><keyword><style  face="normal" font="default" size="100%">Reduced glutathione</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2025</style></year><pub-dates><date><style  face="normal" font="default" size="100%">December 2025</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">17</style></volume><pages><style face="normal" font="default" size="100%">336-341</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Introduction : &lt;/strong&gt;Aluminium is the most abundant metal and the third most common element in the Earth’s crust, following oxygen and silicon. Exposure to aluminium is associated with oxidative damage, primarily due to its ability to disrupt redox balance, generate reactive oxygen species, and impair antioxidant defense mechanisms.This study was aimed to find the potential role of ashwagandha on aluminium induced brain toxicity. &lt;strong&gt;Methods: &lt;/strong&gt;In the present study rats were grouped into 4 groups of 6 rats in each. Brain tissue was removed and processed for biochemical and histopathological analysis. &lt;strong&gt;Results:&lt;/strong&gt; In the present study, administration of aluminium to rats resulted in a significant decrease in tissue GSH levels and a corresponding increase in MDA levels in the aluminium-treated group compared to the normal control.. Treatment with Ashwagandha showed a significant increase in GSH level and decrease in MDA level. Photomicrographic sections of the Brain in Ashwagandha-treated rats showed normla neuronal Count and exposure to Aluminium has caused significant reduction in the neuronal count. Experimental group pretreated with ashwagandha showed a visible increase in neuronal count in different regions of the rat brain.&lt;strong&gt; Conclusion: &lt;/strong&gt;The results revealed that oral administration of aluminium induced adverse oxidative effects in the exposed animals, while treatment with Ashwagandha markedly reduced the extent of aluminium chloride-induced brain injury.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">6</style></issue><work-type><style face="normal" font="default" size="100%">Original Article</style></work-type><section><style face="normal" font="default" size="100%">336</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Anchana Babu&lt;sup&gt;1&lt;/sup&gt;, Rekha D Kini&lt;sup&gt;1*&lt;/sup&gt;, Nayanatara Arun Kumar&lt;sup&gt;1&lt;/sup&gt;, Megha Gokul&lt;sup&gt;1&lt;/sup&gt;, Vandana Blossom&lt;sup&gt;2&lt;/sup&gt;, Sreerag P&lt;sup&gt;3&lt;/sup&gt;, Shymala Nayak&lt;sup&gt;4&lt;/sup&gt; &lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Department of Physiology, Kasturba Medical College Mangalore, Manipal Academy of Higher Education, Manipal, INDIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Department of Anatomy, Kasturba Medical College Mangalore, Manipal Academy of Higher Education, Manipal, INDIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;3&lt;/sup&gt;Department of Physiology, Srinivas Institute of Medical Sciences &amp;amp; Research Centre, Mukka, INDIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;4&lt;/sup&gt;Department of Biochemistry, Kasturba Medical College Mangalore, Manipal Academy of Higher Education, Manipal, INDIA.&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Anchana Babu</style></author><author><style face="normal" font="default" size="100%">Rekha D Kini</style></author><author><style face="normal" font="default" size="100%">Nayanatara Arun Kumar</style></author><author><style face="normal" font="default" size="100%">Megha Gokul</style></author><author><style face="normal" font="default" size="100%">Bhagyalakshmi K</style></author><author><style face="normal" font="default" size="100%">Sneha Shetty B</style></author><author><style face="normal" font="default" size="100%">Vinodini NA</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Potential Role of Ashwagandha [Withania Somnifera] As An Antioxidant On Aluminium Chloride-Induced Testicular Damage In Wistar Rats</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Reactive Oxygen Species</style></keyword><keyword><style  face="normal" font="default" size="100%">Reduced glutathione</style></keyword><keyword><style  face="normal" font="default" size="100%">Sperm morphology</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2025</style></year><pub-dates><date><style  face="normal" font="default" size="100%">September 2025</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">17</style></volume><pages><style face="normal" font="default" size="100%">583-587</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Introduction: &lt;/strong&gt;Aluminium is most prevalent and third most abundant element in earth crust after oxygen. Its exposure in high concentration can accumulate in many organs that damage various organs in living organisms like humans and animals. Although aluminum (Al) is known to induce oxidative damage through various mechanisms including binding to negatively charged phospholipids on the membrane of various tissue cell which are rich in PUFA.Hence,this study was aimed to find the potential role of ashwagandha on aluminum induced testicular toxicity. &lt;strong&gt;Methods: &lt;/strong&gt;Animals were segregated into 4 groups of 6 rats in each. The control group, the Ashwagandha treated group, the Aluminum intoxicated group, pretreated with Ashwagandha with Aluminum intoxicity group. Testicular tissue was removed and were stored in 10% formalin saline and histopathological slides were done . A part of the tissues were processed for estimation of MDA and GSH level. &lt;strong&gt;Results: &lt;/strong&gt;In the present study administration of aluminum in rats showed a significant decrease in the testicular tissue level of GSH and sperm count, as well as increase in the level of MDA and sperm morphology in aluminum treated group compared to normal control. Treatment with Ashwagandha showed a significant increase in testicular GSH level, sperm count and decrease in MDA level sperm morphology. &lt;strong&gt;Conclusion:&lt;/strong&gt; The results of this study revealed that oral Aluminum Chloride administration induced adverse oxidative effects on the exposed animals and treatment with&lt;em&gt; W. somnifera &lt;/em&gt;reduced the extent of aluminium chloride-induced tissue injury&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">5</style></issue><work-type><style face="normal" font="default" size="100%">Original Article</style></work-type><section><style face="normal" font="default" size="100%">583</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Anchana Babu&lt;sup&gt;1&lt;/sup&gt;, Rekha D Kini&lt;sup&gt;1*&lt;/sup&gt;, Nayanatara Arun Kumar&lt;sup&gt;1&lt;/sup&gt;, Megha Gokul&lt;sup&gt;1&lt;/sup&gt;, Bhagyalakshmi K&lt;sup&gt;1&lt;/sup&gt;, Sneha Shetty B&lt;sup&gt;1&lt;/sup&gt;, Vinodini NA&lt;sup&gt;1&lt;/sup&gt; &lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Department of Physiology, Kasturba Medical College Mangalore, Manipal Academy of Higher Education, Manipal, India&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Rahma</style></author><author><style face="normal" font="default" size="100%">Veni Hadju</style></author><author><style face="normal" font="default" size="100%">A. Arsunan Arsin</style></author><author><style face="normal" font="default" size="100%">Aminuddin Syam</style></author><author><style face="normal" font="default" size="100%">Anwar Mallongi</style></author><author><style face="normal" font="default" size="100%">Abd. Farid Lewa</style></author><author><style face="normal" font="default" size="100%">Haerani Harun</style></author><author><style face="normal" font="default" size="100%">Miranti</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">The Effect of Moringa Leaf Extract Intervention Since Preconception Period on the Prevention of Oxidative Stress in Pregnant Women and Adverse Pregnancy Outcomes</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Moringa oleifera</style></keyword><keyword><style  face="normal" font="default" size="100%">Oxidative stress</style></keyword><keyword><style  face="normal" font="default" size="100%">Pregnancy outcomes.</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2023</style></year><pub-dates><date><style  face="normal" font="default" size="100%">April 2023</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">15</style></volume><pages><style face="normal" font="default" size="100%">310-314</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Background:&lt;/strong&gt; Oxidative stress occurs due to an imbalance of oxidants and anti-oxidants and is often associated with poor pregnancy outcomes. Therefore, additional supplementation is needed since the preconception period to prevent oxidative stress and poor pregnancy outcomes. This study aims to examine the effect of supplementing Moringa leaf extract on the prevention of oxidative stress in pregnant women and poor pregnancy outcomes. &lt;strong&gt;Methods: &lt;/strong&gt;This study is an observational study with a prospective cohort study design. The research subjects were third trimester pregnant women consisting of 2 groups, namely the control group who only received Iron-Folic Acid (IFA) supplements (n = 30) and the group that received moringa leaf extract and IFA supplements (n = 26). Then an examination of Malondialdehyde (MDA) levels as a biomarker of oxidative stress was carried out using the ELISA (enzyme-link immune sorbent) method, followed by pregnancy outcomes including birth weight and birth length. Data were analyzed by unpaired t test. &lt;strong&gt;Results: &lt;/strong&gt;The results showed that there were no significant differences in the levels of MDA, birth weight and birth length in the two groups with a p value &amp;gt; 0.05. Nevertheless, MDA level in IFA group was 49.25 nmol/ml higher than Moringa + IFA group which was 47.58 nmol/ml. Likewise, the average of birth weight and birth length in Moringa group were 3042.3 grams and 48.62 cm higher than IFA group which were 2993.7 grams and 48.23 cm. Malondialdehyde levels were also higher in women who had babies with birth weight &amp;lt; 2500 grams was 49.48 nmol/ml and birth length &amp;lt; 48 cm was 49.13 nmol/l compared to birth weight ≥ 2500 grams was 48.50 nmol/ml and birth length ≥ 48 cm was 48.62 nmol/ml but there was no significant difference in the two groups (p&amp;gt;0.05). &lt;strong&gt;Conclusion:&lt;/strong&gt; Oxidative stress in pregnant women tends to be associated with poor pregnancy outcomes. Moringa leaf extract supplementation since preconception can prevent increased oxidative stress and improve pregnancy outcomes.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">2</style></issue><work-type><style face="normal" font="default" size="100%">Original Article </style></work-type><section><style face="normal" font="default" size="100%">310</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Rahma&lt;sup&gt;1,*&lt;/sup&gt;, Veni Hadju&lt;sup&gt;2&lt;/sup&gt;, A. Arsunan Arsin&lt;sup&gt;3&lt;/sup&gt;, Aminuddin Syam&lt;sup&gt;2&lt;/sup&gt;, Anwar Mallongi&lt;sup&gt;4&lt;/sup&gt;, Abd. Farid Lewa&lt;sup&gt;5&lt;/sup&gt;, Haerani Harun&lt;sup&gt;1&lt;/sup&gt;, Miranti&lt;sup&gt;1&lt;/sup&gt;&lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Student of Doctoral Program, Faculty of Public Health, Hasanuddin University, Makassar, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Department of Nutritional Sciences, Faculty of Public Health, Hasanuddin University, Makassar, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;3&lt;/sup&gt;Department of Epidemiology, Faculty of Public Health, Hasanuddin University, Makassar, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;4&lt;/sup&gt;Department of Enviromental Health, Faculty of Public Health, Hasanuddin University, Makassar, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;5&lt;/sup&gt;Departement of Nutrition health, Polytechnic of Palu, INDONESIA.&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Rosmalena</style></author><author><style face="normal" font="default" size="100%">Putu Ayu Widyastuti</style></author><author><style face="normal" font="default" size="100%">Fatmawaty Yazid</style></author><author><style face="normal" font="default" size="100%">Neneng Siti Silfi Ambarwati</style></author><author><style face="normal" font="default" size="100%">Islamudin Ahmad</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Phytochemicals and Antioxidant Activities Evaluation of Origanum vulgare (L.) Stem Bark Extracts</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">2</style></keyword><keyword><style  face="normal" font="default" size="100%">2-diphenyl-1-picrylhydrazyl</style></keyword><keyword><style  face="normal" font="default" size="100%">antioxidant activity</style></keyword><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Origanum vulgare (L.)</style></keyword><keyword><style  face="normal" font="default" size="100%">Phytochemical</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2021</style></year><pub-dates><date><style  face="normal" font="default" size="100%">July 2021</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">13</style></volume><pages><style face="normal" font="default" size="100%">965-970</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;The present study aimed to evaluate phytochemical and antioxidant activity (&lt;em&gt;in vitro&lt;/em&gt; and &lt;em&gt;in vivo&lt;/em&gt;) of &lt;em&gt;Origanum vulgare &lt;/em&gt;(L.) ethanolic extract. The phytochemical test was assessed using the Clule method in ethanol, ethyl acetate, and hexane. &lt;em&gt;In vitro &lt;/em&gt;evaluation of antioxidant activity was determined by radical scavenging assay using DPPH (2,2-diphenyl-1-picrylhydrazyl) as an artificial free radical activity.&lt;em&gt; In vivo &lt;/em&gt;test was conducted to evaluate the effect of malondialdehyde (MDA) level in blood plasma during maximum physical activity treatment.&lt;em&gt; In vivo&lt;/em&gt; test was done using 25 male Sprague Dawley rats in pre and post-test control group design. The phytochemical test of &lt;em&gt;O. vulgare&lt;/em&gt; ethanol extract was showed some compounds, such as a flavonoid, alkaloid, triterpenoid/steroid, essential oil, and tannin, then in ethyl acetate and hexane. &lt;em&gt;In vitro &lt;/em&gt;assay showed that &lt;em&gt;O. vulgare&lt;/em&gt; extract has strong antioxidant activity with an IC&lt;sub&gt;50&lt;/sub&gt; value of 133.47 μg/mL. While in the &lt;em&gt;in vivo&lt;/em&gt; test, the most effective dosage is 20 mg/200 gr B.W., represented by a significant decrease of MDA level (0.509 nmol/mL) before and after treatment. So, the ethanolic extract of clove has potency as an herbal antioxidant because of the low level of IC&lt;sub&gt;50&lt;/sub&gt; and can decrease the MDA level.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">4</style></issue><work-type><style face="normal" font="default" size="100%">Research Article</style></work-type><section><style face="normal" font="default" size="100%">965</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Rosmalena&lt;sup&gt;1&lt;/sup&gt;, Putu Ayu Widyastuti&lt;sup&gt;2&lt;/sup&gt;, Fatmawaty Yazid&lt;sup&gt;1&lt;/sup&gt;, Neneng Siti Silfi Ambarwati&lt;sup&gt;3,&lt;/sup&gt;*, Islamudin Ahmad&lt;sup&gt;4&lt;/sup&gt;&lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Department of Medical Chemistry, Faculty of Medicine, Universitas Indonesia, South Jakarta, 10430 Jakarta, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Medical Student, Faculty of Medicine, Universitas Indonesia, South Jakarta, 10430 Jakarta, INDONESIA&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;3&lt;/sup&gt;Department of Cosmetology, Faculty of Engineering, Universitas Negeri Jakarta, East Jakarta, 13220 Jakarta, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;4&lt;/sup&gt;Department of Pharmaceutical Sciences, Faculty of Pharmacy, Universitas Mulawarman, Samarinda, 75119 East Kalimantan, INDONESIA.&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Rauza Sukma Rita</style></author><author><style face="normal" font="default" size="100%">Elmatris Sy</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Syzygium Cumini Leaves Extract from West Sumatra Indonesia Alleviate Oxidative Stress by Decreasing Malondialdehyde Level and Enhancing Catalase Activity in Rat Induced by Lead Acetate</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Catalase activity</style></keyword><keyword><style  face="normal" font="default" size="100%">Lead acetate</style></keyword><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Rat</style></keyword><keyword><style  face="normal" font="default" size="100%">Syzygium cumini’s leaves</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2021</style></year><pub-dates><date><style  face="normal" font="default" size="100%">November 2021</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">13</style></volume><pages><style face="normal" font="default" size="100%">1408-1412</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Introduction: &lt;/strong&gt;Lead is one of the most dangerous heavy metals in the environment. Contaminated drinking water, battery manufacturing, lead paints, and industrial pollutants are all sources of lead exposure. Lead exposure can cause oxidative stress and is related to many health problems. To prevent oxidative stress caused by lead, the body needs additional antioxidants from the outside body. Syzygium cumini leaf is abundant in antioxidants, which help to minimize oxidative stress caused by lead. &lt;strong&gt;Methods:&lt;/strong&gt; The rats were divided into three groups: negative control, positive control (lead acetate 40 mg/kg BW, 30 days), and treatment (lead acetate 40 mg/kg BW and Syzygium cumini leaves extract 150 mg/kg BW, 30 days). At the end of the experiment, blood was collected and prepared to measure malondialdehyde and catalase activity. &lt;strong&gt;Results&lt;/strong&gt;: The leaf extract of Syzygium cumini reduced serum malondialdehyde levels while increasing catalase activity. Conclusion: Lead exposure induces oxidative stress, which can be reduced by Syzygium cumini’s leaves.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">6</style></issue><work-type><style face="normal" font="default" size="100%">Research Article</style></work-type><section><style face="normal" font="default" size="100%">1408</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Rauza Sukma Rita&lt;sup&gt;1,*&lt;/sup&gt;, Elmatris Sy&lt;sup&gt;2&lt;/sup&gt;&lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Department of Biochemistry, Faculty of Medicine, Universitas Andalas, Padang, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Department of Chemistry, Faculty of Medicine, Universitas Andalas, Padang, INDONESIA&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Tri Widyawati</style></author><author><style face="normal" font="default" size="100%">M Aron Pase</style></author><author><style face="normal" font="default" size="100%">Milahayati Daulay</style></author><author><style face="normal" font="default" size="100%">Imam Bagus Sumantri</style></author><author><style face="normal" font="default" size="100%">Nor Adlin Yusoff</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Evaluation of Myrmecodia pendans Water Extracts on Hematology Profiles, Liver, Kidney Function and Malondialdehyde Level in Healthy Volunteer</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Antioxidant</style></keyword><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Myrmecodia pendans</style></keyword><keyword><style  face="normal" font="default" size="100%">Safety</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2020</style></year><pub-dates><date><style  face="normal" font="default" size="100%">November 2020</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">12</style></volume><pages><style face="normal" font="default" size="100%">1489-1493</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Background: &lt;/strong&gt;Ant Nest (&lt;em&gt;Myrmecodia pendans&lt;/em&gt;) is one of plants that have been used by locals in Indonesia to empirically treat various diseases. Ant Nest&lt;em&gt; in vitro &lt;/em&gt;and&lt;em&gt; in vivo &lt;/em&gt;studies on animals have been widely reported its pharmacological activities as an antioxidant. Unfortunately, scientific proofs reported on this plant as human medicine are still lacking.&lt;strong&gt; Aim: &lt;/strong&gt;This study aimed to ensure effectiveness, and safety due to administration of Ant Nest Water Extract (ANWE) formulated in capsule. &lt;strong&gt;Materials and Methods: &lt;/strong&gt;Twelve volunteers were divided into 2 groups, 6 volunteers in each group. Group I: Ant Nest Water Extract Capsule (ANWEC) in dose of 350 mg ANWE, and group II: Placebo Capsule (PC) which contained 350 mg amylum. Each groups given the capsule once daily for 28 days. The comparison was made between group I and group II. Hematology tests include hemoglobin, leucocyte and thrombocyte. Blood chemistry tests include SGOT, SGPT, total cholesterol, triglycerides, HDL, LDL, glucose, ureum (Ur) and creatinine (Cr). Malondialdehyde (MDA) level was measured at day-28 which was the last day of the intervention. &lt;strong&gt;Results: &lt;/strong&gt;After 28 days administration of ANWE, the SGOT, SGPT, Ur, and Cr showed in normal level as follows SGOT (U/L): 19.2 ± 3.99; SGPT (U/L): 17.2 ± 6.80; Ur (mg/dL): 19.75 ± 3.66; Cr (mg/dL): 1.06 ± 0.13. MDA (ng/mL) level in ANWEC-treated group was significantly lower (117.2±23.8) than PC (147.25±18.7). There was no intolerable complaints during the observation. &lt;strong&gt;Conclusion:&lt;/strong&gt; The study concluded that Water Extract of Ant Nest 350 mg has no damage to liver, kidney and hematology, so it was proven that this plant is safed to be consumed by human for its potency as antioxidant.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">6s</style></issue><work-type><style face="normal" font="default" size="100%">Original Article</style></work-type><section><style face="normal" font="default" size="100%">1489</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Tri Widyawati&lt;sup&gt;1,&lt;/sup&gt;*, M. Aron Pase&lt;sup&gt;2&lt;/sup&gt;, Milahayati Daulay&lt;sup&gt;3&lt;/sup&gt;, Imam Bagus Sumantri&lt;sup&gt;4&lt;/sup&gt;, Nor Adlin Yusoff&lt;sup&gt;5&lt;/sup&gt; &lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Department of Pharmacology and Therapeutic, Faculty of Medicine, Universitas Sumatera Utara, Medan, 20155, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Department of Internal Medicine, Faculty of Medicine, Universitas Sumatera Utara, Medan, 20155, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;3&lt;/sup&gt;Department of Physiology, Faculty of Medicine, Universitas Sumatera Utara, Medan, 20155, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;4&lt;/sup&gt;Department of Pharmacy Biology, Faculty of Pharmacy, Universitas Sumatera Utara, Medan, 20222, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;5&lt;/sup&gt;Integrative Medicine Cluster, Advance Medical and Dental Institute, Universiti Sains Malaysia, Penang, 13200, MALAYSIA.&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Fernández-Flores N</style></author><author><style face="normal" font="default" size="100%">Rojas-Cardenas NF</style></author><author><style face="normal" font="default" size="100%">Vásquez-Quispe AD</style></author><author><style face="normal" font="default" size="100%">Chávez-Flores Juana E</style></author><author><style face="normal" font="default" size="100%">Justil-Guerrero Hugo J</style></author><author><style face="normal" font="default" size="100%">Parreño-Tipian JM</style></author><author><style face="normal" font="default" size="100%">Silva-Correa Carmen R</style></author><author><style face="normal" font="default" size="100%">Villarreal-La Torre Víctor E</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Protection of Erythrocytes against Lipoperoxidation and Antiinflammatory Effects of Ethanolic Extract of Encelia canescens Lam Leaves in Mice</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Antiinflammatory</style></keyword><keyword><style  face="normal" font="default" size="100%">Carrageenan</style></keyword><keyword><style  face="normal" font="default" size="100%">Encelia canescens Lam</style></keyword><keyword><style  face="normal" font="default" size="100%">Hemolysis</style></keyword><keyword><style  face="normal" font="default" size="100%">Lipoperoxidation</style></keyword><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2020</style></year><pub-dates><date><style  face="normal" font="default" size="100%">June 2020</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">12</style></volume><pages><style face="normal" font="default" size="100%">798-804</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Background:&lt;/strong&gt; &lt;em&gt;Encelia canescens&lt;/em&gt; Lam is a plant traditionally used in Peru for medicinal purposes, and is attributed antioxidant properties, indicating that it could be used in the prevention of non-communicable diseases. &lt;strong&gt;Objective:&lt;/strong&gt; This study aims to evaluate the protection of erythrocytes from lipoperoxidation and the anti-inflammatory effect of ethanolic extract of &lt;em&gt;E. canescens&lt;/em&gt; leaves in mice. &lt;strong&gt;Materials and methods: &lt;/strong&gt;Protection from lipoperoxidation was evaluated by inhibition of hemolysis and quantifying malondialdehyde (MDA) concentration against oxidative stress induced with hydrogen peroxide (H&lt;sub&gt;2&lt;/sub&gt;O&lt;sub&gt;2&lt;/sub&gt;) at 200, 150, 100, 50 and 25 μg/mL &lt;em&gt;E. canescens&lt;/em&gt; concentrations. The 1% carrageenan-induced air pouch model was used for evaluated inflammation, where albumin, total proteins, MDA, number and leukocyte differentiation were determined in the exudate, and a histopathological evaluation was performed. The concentrations evaluated were 100, 250 and 500 mg/kg of &lt;em&gt;E. canescens&lt;/em&gt;&lt;strong&gt; Results:&lt;/strong&gt; All the concentrations evaluated protected protected erythrocytes from lipoperoxidation (&lt;em&gt;p&lt;/em&gt;&amp;lt;0.05), being E.D. value 200 μg/mL. Regarding anti-inflammatory effect, the albumin, total proteins and MDA values of the treatment groups were lower than carrageenan 1% group (&lt;em&gt;p&lt;/em&gt;&amp;lt;0.05), but, due to less leukocyte migration and presence of macrophages and the histopathological evaluation, the E.D value was 500 mg/kg. &lt;strong&gt;Conclusion:&lt;/strong&gt; Ethanolic extracts of &lt;em&gt;E. canescens &lt;/em&gt;leaves protect erythrocytes from lipoperoxidation and have dose-dependent anti-inflammatory effects maybe for presence of p-hydroxyacetophenone-derived, and these could be new safer anti-inflammatories.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">4</style></issue><work-type><style face="normal" font="default" size="100%">Original Article</style></work-type><section><style face="normal" font="default" size="100%">798</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Fernández-Flores N&lt;sup&gt;1&lt;/sup&gt;, Rojas- Cardenas NF&lt;sup&gt;1&lt;/sup&gt;, Vásquez-Quispe AD&lt;sup&gt;1&lt;/sup&gt;, Chávez-Flores Juana E&lt;sup&gt;1,&lt;/sup&gt;*, Justil-Guerrero Hugo J&lt;sup&gt;1&lt;/sup&gt;, Parreño- Tipian JM&lt;sup&gt;1&lt;/sup&gt;, Silva-Correa Carmen R&lt;sup&gt;2&lt;/sup&gt;, Villarreal-La Torre Víctor E&lt;sup&gt;2,&lt;/sup&gt;* &lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Facultad de Farmacia y Bioquímica, Universidad Norbert Wiener, PERÚ.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Facultad de Farmacia y Bioquímica, Universidad Nacional de Trujillo, PERÚ.&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Abdul Gofur</style></author><author><style face="normal" font="default" size="100%">Agung Witjoro</style></author><author><style face="normal" font="default" size="100%">Erni Widya Ningtiyas</style></author><author><style face="normal" font="default" size="100%">Evi Setyowati</style></author><author><style face="normal" font="default" size="100%">Siti Aminatul Mukharromah</style></author><author><style face="normal" font="default" size="100%">Mochammad Fitri Atho’illah</style></author><author><style face="normal" font="default" size="100%">Sri Rahayu Lestari</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">The Evaluation of Dietary Black Soybean and Purple Sweet Potato on Insulin Sensitivity in Streptozotocin - Induced Diabetic Rats</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Antioxidant</style></keyword><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Necrosis</style></keyword><keyword><style  face="normal" font="default" size="100%">Superoxide dismutase</style></keyword><keyword><style  face="normal" font="default" size="100%">T2DM</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2019</style></year><pub-dates><date><style  face="normal" font="default" size="100%">July 2019</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">11</style></volume><pages><style face="normal" font="default" size="100%">639-646</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Introduction: &lt;/strong&gt;Hyperglycemia mediates the production of excess free radicals and reduces endogenous antioxidant in type 2 diabetes mellitus (T2DM). Black soybean (BSB) is rich in antioxidant mainly from isoflavones, whereas the antioxidant of purple sweet potato (PSP) comes from high anthocyanin. The study aimed to evaluate the efficacy of BSB, PSP, and its combination on malondialdehyde (MDA), superoxide dismutase (SOD) concentrations, insulin and insulin receptor substrate-1 (IRS-1) expression in T2DM rats. &lt;strong&gt;Methods: &lt;/strong&gt;T2DM induced by high-calorie diet for five weeks and then injected with a low dose of streptozotocin (30 mg/kg BW) intraperitoneally. The DM rats then treated with black soybean (DM + BSB), purple sweet potato (DM + PSP) and the combination of BSB and PSP 1:3, 2:2, 3:1 respectively (DM + C1- 3). Treatments were given for thirty days. The effect of BSB, PSP and its combination evaluated by measuring SOD and MDA, necrosis in pancreas evaluated through hematoxylin-eosin (HE) and insulin and IRS-1 expression in pancreas through immunohistochemistry-fluorescence (IHC-F). &lt;strong&gt;Results: &lt;/strong&gt;Our result indicated that there were no significant differences of BSB and the combination in decreasing MDA concentrations. The ratio of BSB and PSP combination of 2:2 increase SOD towards near normal, decrease necrosis, and improve insulin and IRS-1. &lt;strong&gt;Conclusion: &lt;/strong&gt;The combination of BSB and PSP had the potential to improve insulin sensitivity through the increase of SOD, reduce necrosis, and improve insulin and IRS-1 expression.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">4</style></issue><work-type><style face="normal" font="default" size="100%">Original Article</style></work-type><section><style face="normal" font="default" size="100%">639</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p class=&quot;rtejustify&quot;&gt;&lt;strong&gt;Abdul Gofur&lt;sup&gt;1,*&lt;/sup&gt;, Agung Witjoro&lt;sup&gt;1&lt;/sup&gt;, Erni Widya Ningtiyas&lt;sup&gt;1&lt;/sup&gt;, Evi Setyowati&lt;sup&gt;1&lt;/sup&gt;, Siti Aminatul Mukharromah&lt;sup&gt;1&lt;/sup&gt;, Mochammad Fitri Atho’illah&lt;sup&gt;2&lt;/sup&gt;, Sri Rahayu Lestari&lt;sup&gt;1&lt;/sup&gt; &lt;/strong&gt;&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;1&lt;/sup&gt;Department of Biology, Faculty of Mathematics and Natural Science, Universitas Negeri Malang, Jl. Semarang No. 5, Malang 65145, INDONESIA.&lt;/p&gt;

&lt;p class=&quot;rtejustify&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Department of Biology, Faculty of Mathematics and Natural Science, Brawijaya University, Jl. Veteran, Malang 65145, INDONESIA.&lt;/p&gt;
</style></auth-address></record><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Ladachart Taepongsorat</style></author><author><style face="normal" font="default" size="100%">Methin Phadungkit</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Effects of Asparagus racemosus Root Extracts on Serum Lipid Profiles, Lipid Peroxidation and Superoxide Dismutase in Ovariectomized Rat</style></title><secondary-title><style face="normal" font="default" size="100%">Pharmacognosy Journal</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Asparagus racemosus</style></keyword><keyword><style  face="normal" font="default" size="100%">Lipid profiles</style></keyword><keyword><style  face="normal" font="default" size="100%">Malondialdehyde</style></keyword><keyword><style  face="normal" font="default" size="100%">Ovariectomized rat</style></keyword><keyword><style  face="normal" font="default" size="100%">Superoxide dismutase</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2018</style></year><pub-dates><date><style  face="normal" font="default" size="100%">August 2018</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">10</style></volume><pages><style face="normal" font="default" size="100%">1036-1041</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p style=&quot;text-align: justify;&quot;&gt;&lt;strong&gt;Background:&lt;/strong&gt; Once rats have been ovariectomized they have a high risk of cardiovascular disease due to changes in the blood cholesterol and lipid profile. &lt;strong&gt;Objective:&lt;/strong&gt; To investigate the effects of &lt;em&gt;Asparagus racemosus&lt;/em&gt; (AR) root extract on the serum lipid profiles, lipid peroxidation and antioxidant levels in ovariectomized rats. &lt;strong&gt;Methods:&lt;/strong&gt; Twenty-five, two month-old female Wistar rats were randomly divided into five groups: SH, OVX,OEE, OAAR and OEAR. The daily doses of 500 mg/KgBW of the AR root extracts for five weeks.The levels of serum TG, TC, HDL, LDL, the liver, kidney and uterine tissue lipid peroxidation and SOD levels were determined. &lt;strong&gt;Results:&lt;/strong&gt; Serum TC and LDL showed no significant differences in any groups. Serum TG of the OAAR and OEAR groups were not significantly different. The serum HDL of the OAAR and OEAR groups were significantly lower than the OEE group. The liver MDA levels of the OAAR and OEAR groups were significantly decreased compared with the OVX and OEE groups while the SOD level of the OAAR group was significantly increased. The MDA levels in the kidney and uterine of the treated group showed no significant difference. The SOD levels in the kidney of the treated group were not different but the SOD levels in uterine were significantly decreased. &lt;strong&gt;Conclusion:&lt;/strong&gt; It can be believed that the lipid profiles were maybe regulated via estrogen.The AR extract has low effects on the lipid profiles at this dose and duration of treatment. The capacity of the extracts to decrease the MDA level and increase the SOD level in this study clearly reflected the antioxidant efficiency of these substances.&lt;/p&gt;</style></abstract><issue><style face="normal" font="default" size="100%">5</style></issue><work-type><style face="normal" font="default" size="100%">Original Article</style></work-type><section><style face="normal" font="default" size="100%">1036</style></section><auth-address><style face="normal" font="default" size="100%">&lt;p style=&quot;text-align: justify;&quot;&gt;&lt;strong&gt;Ladachart Taepongsorat&lt;sup&gt;1*&lt;/sup&gt;, Methin Phadungkit&lt;sup&gt;2&lt;/sup&gt;&lt;/strong&gt;&lt;/p&gt;
&lt;p style=&quot;text-align: justify;&quot;&gt;&lt;sup&gt; 1&lt;/sup&gt;Faculty of Medicine, Mahasarakham University, Mahasarakham 44000, THAILAND.&lt;/p&gt;
&lt;p style=&quot;text-align: justify;&quot;&gt;&lt;sup&gt;2&lt;/sup&gt;Pharmaceutical Chemistry and Natural Products Research Unit, Faculty of Pharmacy, Mahasarakham University, Mahasarakham 44150, THAILAND.&lt;/p&gt;</style></auth-address></record></records></xml>