Traumatic Brain Injury

Pharmacognosy Journal (Phcog J.) covers different topics in natural product drug discovery, and also publishes manuscripts that describe pharmacognostic investigations, evaluation reports, methods, techniques and applications of all forms of medicinal plant research
Distinctions: The most widely read, cited, and known Pharmacognosy journal and website is well browsed with all the articles published. More than 50,000 readers in nearly every country in the world each month
ISSN : 0975-3575 ; Frequency : Rapid at a time publication (6 issues/year)
Indexed and Abstracted in : SCOPUS, Scimago Journal Ranking, Chemical Abstracts, Excerpta Medica / EMBASE, Google Scholar, CABI Full Text, Index Copernicus, Ulrich’s International Periodical Directory, ProQuest, Journalseek & Genamics, PhcogBase, EBSCOHost, Academic Search Complete, Open J-Gate, SciACCESS.
Rapid publication: Average time from submission to first decision is 30 days and from acceptance to In Press online publication is 45 days.
Open Access Journal: Pharmacognosy Journal is an open access journal, which allows authors to fund their article to be open access from publication.

Relationship Between Cerebrospinal Fluid S100B Levels with Glasgow Coma Scale and Rotterdam CT Score in Traumatic Brain Injury Patients

The Role of Neuron Specific Enolase, S100B, Glial Fibrillary Acidic Protein, and Myelin Basic Protein as Prognostic and Survival Values in Traumatic Brain Injury: Systematic Review and Meta-analysis

The Impact of Kaempferia galanga L. Extract on Nerve Growth Factor Expression in A Rat Model of Traumatic Brain Injury

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Traumatic Brain Injury

Relationship Between Cerebrospinal Fluid S100B Levels with Glasgow Coma Scale and Rotterdam CT Score in Traumatic Brain Injury Patients

The Role of Neuron Specific Enolase, S100B, Glial Fibrillary Acidic Protein, and Myelin Basic Protein as Prognostic and Survival Values in Traumatic Brain Injury: Systematic Review and Meta-analysis

The Impact of Kaempferia galanga L. Extract on Nerve Growth Factor Expression in A Rat Model of Traumatic Brain Injury